The content you've requested is available without charge only to active Sigma Xi members and American Scientist subscribers.
If you are an active member or an individual subscriber, please log in now in order to access this article.
If you are not a member or individual subscriber, you can:
The number of people in the United States affected by this neurodegenerative disorder is expected to reach 16 million by 2050, with no useful medical therapies in sight. Two current theories about the disease’s etiology involve tissue disorders observed in patients: amyloid plaques and neurofibrillary tangles. The author, however, proposes that these are secondary conditions brought about by a decline in neuroangiogenesis—reduced formation of capillaries in certain parts of the brain that leads to plaques and tangles. The neuroangiogenesis hypothesis for Alzheimer’s and other forms of cognitive decline with aging suggests therapies that increase vascularization, and some of these approaches have already been used successfully in animals with brain injuries.
Connect With Us:
An early peek at each new issue, with descriptions of feature articles, columns, and more. Every other issue contains links to everything in the latest issue's table of contents.News of book reviews published in American Scientist and around the web, as well as other noteworthy happenings in the world of science books.
To sign up for automatic emails of the American Scientist Update and Scientists' Nightstand issues, create an online profile, then sign up in the My AmSci area.
JSTOR, the online academic archive, contains complete back issues of American Scientist from 1913 (known then as the Sigma Xi Quarterly) through 2005.
The table of contents for each issue is freely available to all users; those with institutional access can read each complete issue.
View the full collection here.
Receive notification when new content is posted from the entire website, or choose from the customized feeds available.